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Is the nap zone controlled by a ligh...

Krupa, Susanne.

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Is the nap zone controlled by a light-sensitive circadian arousal process?

紀錄類型:	書目-語言資料,印刷品 : Monograph/item
正題名/作者:	Is the nap zone controlled by a light-sensitive circadian arousal process?/
作者:	Krupa, Susanne.
面頁冊數:	207 p.
附註:	Adviser: Roger Broughton.
Contained By:	Dissertation Abstracts International65-03B.
標題:	Psychology, Physiological. -
電子資源:	http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=NQ90000
ISBN:	9780612900004

Is the nap zone controlled by a light-sensitive circadian arousal process?
Krupa, Susanne.

Is the nap zone controlled by a light-sensitive circadian arousal process? - 207 p.

Adviser: Roger Broughton.

Thesis (Ph.D.)--University of Ottawa (Canada), 2003.

This study attempts to identify the mechanism of the so-called afternoon nap zone. More specifically, it investigates a recent proposal that the afternoon nap zone represents the point in time after morning awakening when process-S has increased to a sufficiently high level to facilitate sleep onset, while at the same time an SCN-controlled circadian arousal process has not yet risen high enough to reverse this increased sleepiness.

ISBN: 9780612900004Subjects--Topical Terms:

1017869
Psychology, Physiological.

Is the nap zone controlled by a light-sensitive circadian arousal process?
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245 1 0 $a Is the nap zone controlled by a light-sensitive circadian arousal process?
300 $a 207 p.
500 $a Adviser: Roger Broughton.
500 $a Source: Dissertation Abstracts International, Volume: 65-03, Section: B, page: 1591.
502 $a Thesis (Ph.D.)--University of Ottawa (Canada), 2003.
520 $a This study attempts to identify the mechanism of the so-called afternoon nap zone. More specifically, it investigates a recent proposal that the afternoon nap zone represents the point in time after morning awakening when process-S has increased to a sufficiently high level to facilitate sleep onset, while at the same time an SCN-controlled circadian arousal process has not yet risen high enough to reverse this increased sleepiness.
520 $a Eight normal male subjects aged 20--30 years, were monitored on two separate occasions under low ambient light (150 lux). Night sleep hours were maintained at 23:00--06:00h. PSG monitoring included EEG (C3-A2, O2-A1), right EOG-M1, left EOG-M2, submental EMG and core body temperature recorded continuously by a combination of the Oxford Medilog 9000 8-channel ambulatory recorder and the Minilogger temperature monitoring system. Following a baseline 24-hour day, bright light stimulation (10,000 lux) was given on two consecutive days either in the evening (20:00--22:00h) or morning (06:00--08:00h) in counter-balanced fashion with a 30 day washout periods between. Other than during a period of bright light stimulation, the level of daytime arousal was assessed every 60 min by quantified EEG spectral power followed by a 10 min duration simple reaction time test.
520 $a In the baseline condition both the performance and Q-EEG variables confirmed the presence of a transitory afternoon nap zone as indexed by the timing of poorest performance and of greatest spectral power in a number of Q-EEG measures. Evening bright light treatment phase delayed these nap zone measures. Conversely, morning bright light phase advanced these measures.
520 $a The finding that the timing of poorest performance and of the Q-EEG determined nap zone can be phase delayed by evening light and phase advanced by morning light supports the hypothesis tested, as well as supporting the existence of an SCN-dependent circadian arousal system in humans similar to that described by Edgar et al. (1993) in sub-human primates.
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