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An evaluation of neuromechanical dec...

Needle, Alan R.

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An evaluation of neuromechanical decoupling following ankle joint injury.

紀錄類型:	書目-語言資料,印刷品 : Monograph/item
正題名/作者:	An evaluation of neuromechanical decoupling following ankle joint injury./
作者:	Needle, Alan R.
面頁冊數:	201 p.
附註:	Source: Dissertation Abstracts International, Volume: 75-02(E), Section: B.
Contained By:	Dissertation Abstracts International75-02B(E).
標題:	Health Sciences, Recreation. -
電子資源:	http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=3598723
ISBN:	9781303475184

An evaluation of neuromechanical decoupling following ankle joint injury.
Needle, Alan R.

An evaluation of neuromechanical decoupling following ankle joint injury. - 201 p.

Source: Dissertation Abstracts International, Volume: 75-02(E), Section: B.

Thesis (Ph.D.)--University of Delaware, 2013.

Ankle sprains occur in nearly 50 percent of the population, with close to 70 percent of patients experiencing residual symptoms or recurrent injury. Joint laxity and neurological function have been proposed to change following ligamentous injury; however, few studies have investigated these measures concurrently using direct methodology. As a result, it is unclear if neurological function may no longer be appropriately tuned to mechanical restraints at the joint among patients with functional ankle instability. Furthermore it remains unknown what adaptations allow some patients to "cope" and avoid this neuromechanical decoupling related to instability after injury. The purpose of this study was, therefore, to evaluate the relationships between joint laxity & stiffness, and neurological function as measured through muscle spindle activity, somatosensory cortex activation, corticospinal excitability, and motoneuronal pool excitability, among subjects with and without functional ankle instability. The results of this study indicate neuromechanical coupling is a factor necessary for maintaining joint stability, as evident through associations between greater joint laxity and higher muscle spindle activity, cortical activation, and greater tibialis anterior excitability. Relationships were also observed among neurological factors, where greater corticospinal excitability correlated with higher muscle spindle activity, but less cortical activation. Our results suggest some neuromechanical decoupling occurs in patients with functionally unstable ankles because they exhibited greater joint laxity without associated changes in neurophysiologic variables, indicating the nervous system may have failed to adapt despite altered mechanical function. Copers, however, demonstrated potential enhancements in coupling, where cortical excitability was better tuned to the laxity and sensation of the joint, as well as demonstrating reflexive inhibition throughout joint loading. These findings offer new insights into the etiology of ankle instability and support treatment of initial injury to occur on an individualized basis.

ISBN: 9781303475184Subjects--Topical Terms:

1018003
Health Sciences, Recreation.

An evaluation of neuromechanical decoupling following ankle joint injury.
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502 $a Thesis (Ph.D.)--University of Delaware, 2013.
520 $a Ankle sprains occur in nearly 50 percent of the population, with close to 70 percent of patients experiencing residual symptoms or recurrent injury. Joint laxity and neurological function have been proposed to change following ligamentous injury; however, few studies have investigated these measures concurrently using direct methodology. As a result, it is unclear if neurological function may no longer be appropriately tuned to mechanical restraints at the joint among patients with functional ankle instability. Furthermore it remains unknown what adaptations allow some patients to "cope" and avoid this neuromechanical decoupling related to instability after injury. The purpose of this study was, therefore, to evaluate the relationships between joint laxity & stiffness, and neurological function as measured through muscle spindle activity, somatosensory cortex activation, corticospinal excitability, and motoneuronal pool excitability, among subjects with and without functional ankle instability. The results of this study indicate neuromechanical coupling is a factor necessary for maintaining joint stability, as evident through associations between greater joint laxity and higher muscle spindle activity, cortical activation, and greater tibialis anterior excitability. Relationships were also observed among neurological factors, where greater corticospinal excitability correlated with higher muscle spindle activity, but less cortical activation. Our results suggest some neuromechanical decoupling occurs in patients with functionally unstable ankles because they exhibited greater joint laxity without associated changes in neurophysiologic variables, indicating the nervous system may have failed to adapt despite altered mechanical function. Copers, however, demonstrated potential enhancements in coupling, where cortical excitability was better tuned to the laxity and sensation of the joint, as well as demonstrating reflexive inhibition throughout joint loading. These findings offer new insights into the etiology of ankle instability and support treatment of initial injury to occur on an individualized basis.
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