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Effects of visual cortex lesions on ...
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Sonnenberg, Douglas C.
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Effects of visual cortex lesions on modulation of the cutaneous and acoustic blink reflexes and choice reaction time.
紀錄類型:
書目-電子資源 : Monograph/item
正題名/作者:
Effects of visual cortex lesions on modulation of the cutaneous and acoustic blink reflexes and choice reaction time./
作者:
Sonnenberg, Douglas C.
面頁冊數:
39 p.
附註:
Source: Masters Abstracts International, Volume: 42-04, page: 1401.
Contained By:
Masters Abstracts International42-04.
標題:
Psychology, Experimental. -
電子資源:
http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=1418065
ISBN:
0496228463
Effects of visual cortex lesions on modulation of the cutaneous and acoustic blink reflexes and choice reaction time.
Sonnenberg, Douglas C.
Effects of visual cortex lesions on modulation of the cutaneous and acoustic blink reflexes and choice reaction time.
- 39 p.
Source: Masters Abstracts International, Volume: 42-04, page: 1401.
Thesis (M.A.)--University of Missouri - Columbia, 2003.
Weak visual prestimulation has been shown to facilitate the blink reflex at short and long lead intervals but to inhibit it at intermediate intervals. Blink inhibition at these intermediate lead intervals has generally been attributed to pathways that are entirely subcortical. However, the present study of 13 patients with unilateral visual cortex damage showed that presentation of a prepulse to the blind hemifield was not sufficient to inhibit the blink reflex to cutaneous or acoustic reflexogenic stimuli. The blind hemifield prepulses also failed to facilitate reflexes at short and long lead intervals or to speed voluntary reactions to the reflexogenic stimuli. These results imply that reflex modulation by a visual prepulse is mediated by visual cortex.
ISBN: 0496228463Subjects--Topical Terms:
517106
Psychology, Experimental.
Effects of visual cortex lesions on modulation of the cutaneous and acoustic blink reflexes and choice reaction time.
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Weak visual prestimulation has been shown to facilitate the blink reflex at short and long lead intervals but to inhibit it at intermediate intervals. Blink inhibition at these intermediate lead intervals has generally been attributed to pathways that are entirely subcortical. However, the present study of 13 patients with unilateral visual cortex damage showed that presentation of a prepulse to the blind hemifield was not sufficient to inhibit the blink reflex to cutaneous or acoustic reflexogenic stimuli. The blind hemifield prepulses also failed to facilitate reflexes at short and long lead intervals or to speed voluntary reactions to the reflexogenic stimuli. These results imply that reflex modulation by a visual prepulse is mediated by visual cortex.
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http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=1418065
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