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Role of endothelin in modulating chr...

Abram, Sean R.

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Role of endothelin in modulating chronic pressure-natriuresis and blood pressure regulation in response to changes in dietary sodium intake.

Record Type:	Electronic resources : Monograph/item
Title/Author:	Role of endothelin in modulating chronic pressure-natriuresis and blood pressure regulation in response to changes in dietary sodium intake./
Author:	Abram, Sean R.
Description:	109 p.
Notes:	Source: Dissertation Abstracts International, Volume: 65-06, Section: B, page: 2722.
Contained By:	Dissertation Abstracts International65-06B.
Subject:	Biology, Animal Physiology. -
Online resource:	http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=3135855
ISBN:	0496830589

Role of endothelin in modulating chronic pressure-natriuresis and blood pressure regulation in response to changes in dietary sodium intake.
Abram, Sean R.

Role of endothelin in modulating chronic pressure-natriuresis and blood pressure regulation in response to changes in dietary sodium intake. - 109 p.

Source: Dissertation Abstracts International, Volume: 65-06, Section: B, page: 2722.

Thesis (Ph.D.)--The University of Mississippi Medical Center, 2004.

Since its discovery in 1988, much has been learned about the role endothelin (ET or ET-1) in the pathophysiology of hypertension. Aberrations in renal ET-1 expression, synthesis, and excretion are now commonly associated with the hypertensive st

ISBN: 0496830589Subjects--Topical Terms:

1017835
Biology, Animal Physiology.

Role of endothelin in modulating chronic pressure-natriuresis and blood pressure regulation in response to changes in dietary sodium intake.
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040 $a UnM $c UnM
100 1 $a Abram, Sean R. $3 1933946
245 1 0 $a Role of endothelin in modulating chronic pressure-natriuresis and blood pressure regulation in response to changes in dietary sodium intake.
300 $a 109 p.
500 $a Source: Dissertation Abstracts International, Volume: 65-06, Section: B, page: 2722.
500 $a Adviser: John E. Hall.
502 $a Thesis (Ph.D.)--The University of Mississippi Medical Center, 2004.
520 $a Since its discovery in 1988, much has been learned about the role endothelin (ET or ET-1) in the pathophysiology of hypertension. Aberrations in renal ET-1 expression, synthesis, and excretion are now commonly associated with the hypertensive st
520 $a The overall goal of this dissertation was to test the central hypothesis that chronic blockade of ETB receptors causes salt-sensitive hypertension by impairing renal-pressure natriuresis. To achieve this goal, the ETB receptor was blocked using
520 $a The first important finding was the fact that the renal endothelin system is upregulated in response to increases in sodium intake. Secondly, blockade of the ETB receptors results in a rightward shift of the chronic pressure-natriuresis relation
590 $a School code: 0805.
650 4 $a Biology, Animal Physiology. $3 1017835
650 4 $a Health Sciences, Medicine and Surgery. $3 1017756
650 4 $a Biophysics, Medical. $3 1017681
650 4 $a Health Sciences, Pathology. $3 1017854
690 $a 0433
690 $a 0564
690 $a 0760
690 $a 0571
710 2 0 $a The University of Mississippi Medical Center. $3 1017852
773 0 $t Dissertation Abstracts International $g 65-06B.
790 1 0 $a Hall, John E., $e advisor
790 $a 0805
791 $a Ph.D.
792 $a 2004
856 4 0 $u http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=3135855