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Central versus peripheral limitation...
~
Vella, Chantal Anne.
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Central versus peripheral limitations to maximal oxygen uptake.
Record Type:
Electronic resources : Monograph/item
Title/Author:
Central versus peripheral limitations to maximal oxygen uptake./
Author:
Vella, Chantal Anne.
Description:
109 p.
Notes:
Source: Dissertation Abstracts International, Volume: 65-04, Section: B, page: 1823.
Contained By:
Dissertation Abstracts International65-04B.
Subject:
Health Sciences, Recreation. -
Online resource:
http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=3129658
ISBN:
0496769162
Central versus peripheral limitations to maximal oxygen uptake.
Vella, Chantal Anne.
Central versus peripheral limitations to maximal oxygen uptake.
- 109 p.
Source: Dissertation Abstracts International, Volume: 65-04, Section: B, page: 1823.
Thesis (Ph.D.)--The University of New Mexico, 2004.
The purposes of this dissertation were to quantify the central cardiovascular responses to incremental exercise and examine the cardiovascular data at maximal exercise for the presence of a plateau (Study A), and to investigate the VO 2VENT across a range of ventilations representing an incremental exercise test to VO2max (Study B).
ISBN: 0496769162Subjects--Topical Terms:
1018003
Health Sciences, Recreation.
Central versus peripheral limitations to maximal oxygen uptake.
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Central versus peripheral limitations to maximal oxygen uptake.
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109 p.
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Source: Dissertation Abstracts International, Volume: 65-04, Section: B, page: 1823.
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Chair: Rob Robergs.
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Thesis (Ph.D.)--The University of New Mexico, 2004.
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The purposes of this dissertation were to quantify the central cardiovascular responses to incremental exercise and examine the cardiovascular data at maximal exercise for the presence of a plateau (Study A), and to investigate the VO 2VENT across a range of ventilations representing an incremental exercise test to VO2max (Study B).
520
$a
Twenty-one endurance-trained athletes performed an initial VO2max test, followed by two incremental exercise tests to VO2max, two incremental exercise tests to 90% of VO2max, and one hyperventilation trial.
520
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Study A. Cardiac output was indirectly measured using the exponential and equilibrium CO2 rebreathing methods. The stroke volume and a-VO2 difference were calculated using standard equations. Regression analyses were performed to describe the relationships of the cardiovascular variables with VO2 during incremental exercise to VO2max. Significant nonlinear relationships were found between all cardiovascular variables and VO2. A plateau in cardiac output at VO2max was identified in three subjects. Stroke volume plateaued at an average of 37.0 +/- 12.5% of VO2max in 18 subjects and increased continuously to VO2max in three subjects. The a-VO2 difference progressively increased to VO2max in 17 subjects and revealed a plateau response in four subjects. Our data support the theories that, depending on the person, either of central or peripheral factors could be more influential in limiting VO2max.
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Study B. Subjects were asked to mimic nine different VE volumes corresponding to their exercise VE during the initial VO2max test. Regression analysis was used to determine the relationship between VE and VO2VENT. Whole body VO2 was then corrected for the VO2VENT to determine if of the incidence of a plateau in VO2 at maximal exercise was greater when the whole body VO2 is corrected for the VO2VENT.
520
$a
The maximal VO2VENT equaled 8.8% +/- 3.3% of VO 2max and ranged from 5.0--17.6%. VO2VENT increased exponentially with increasing VE, but there was considerable subject variability in the oxygen cost per liter of VE as VE increased. Subtracting the VO2VENT from whole body VO2 increased the detection of a plateau at VO2 max. Our data indicate that the VO2VENT is a significant portion of VO2max and may limit maximal exercise performance.
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School code: 0142.
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The University of New Mexico.
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http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=3129658
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