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Acute neuronal injury = the role of ...

Fujikawa, Denson G.

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Acute neuronal injury = the role of excitotoxic programmed cell death mechanisms /

Record Type:	Electronic resources : Monograph/item
Title/Author:	Acute neuronal injury/ edited by Denson G. Fujikawa.
Reminder of title:	the role of excitotoxic programmed cell death mechanisms /
other author:	Fujikawa, Denson G.
Published:	Cham :Springer International Publishing : : 2018.,
Description:	xii, 215 p. :ill., digital ;24 cm.
[NT 15003449]:	Introduction -- Excitotoxic programmed cell death involves caspase-independent mechanisms -- To survive or to die: how neurons deal with it -- Oxidative damage mechanisms in traumatic brain injury and antioxidant neuroprotective approaches -- Mitochondrial damage in traumatic CNS injury -- Neuroprotective agents target molecular mechanisms of programmed cell death after traumatic brain injury -- Involvement of apoptosis-inducing factor (AIF) in neuronal death following cerebral ischemia -- Apoptosis-inducing factor translocation to nuclei after transient global ischemia -- Necroptosis in cerebral ischemia -- Histological and elemental changes in ischemic stroke -- Hypoglycemic brain damage -- Activation of caspase-independent programmed pathways in seizure-induced neuronal necrosis -- Conclusion.
Contained By:	Springer eBooks
Subject:	Nervous system - Diseases -
Online resource:	http://dx.doi.org/10.1007/978-3-319-77495-4
ISBN:	9783319774954

Acute neuronal injury = the role of excitotoxic programmed cell death mechanisms /
Acute neuronal injurythe role of excitotoxic programmed cell death mechanisms /[electronic resource] :edited by Denson G. Fujikawa. - 2nd ed. - Cham :Springer International Publishing :2018. - xii, 215 p. :ill., digital ;24 cm.

Introduction -- Excitotoxic programmed cell death involves caspase-independent mechanisms -- To survive or to die: how neurons deal with it -- Oxidative damage mechanisms in traumatic brain injury and antioxidant neuroprotective approaches -- Mitochondrial damage in traumatic CNS injury -- Neuroprotective agents target molecular mechanisms of programmed cell death after traumatic brain injury -- Involvement of apoptosis-inducing factor (AIF) in neuronal death following cerebral ischemia -- Apoptosis-inducing factor translocation to nuclei after transient global ischemia -- Necroptosis in cerebral ischemia -- Histological and elemental changes in ischemic stroke -- Hypoglycemic brain damage -- Activation of caspase-independent programmed pathways in seizure-induced neuronal necrosis -- Conclusion.

An overview of the biochemical mechanisms that produce acute nerve cell death in the brain. Covers injuries and disorders including stroke, brain and spinal cord trauma, hypoglycemic coma, and prolonged epileptic seizures. All of these lead to high concentrations of calcium in nerve cells which, in turn, causes degradation of cytoplasmic proteins, cleavage of nuclear DNA, and eventually cell death. The Second Edition contains 11 thoroughly updated chapters and 3 additional chapters that did not appear in the previous edition.

ISBN: 9783319774954

Standard No.: 10.1007/978-3-319-77495-4doiSubjects--Topical Terms:

1066203
Nervous system
--Diseases

LC Class. No.: RC347 / .A288 2018

Dewey Class. No.: 616.807

Acute neuronal injury = the role of excitotoxic programmed cell death mechanisms /
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245 0 0 $a Acute neuronal injury $h [electronic resource] : $b the role of excitotoxic programmed cell death mechanisms / $c edited by Denson G. Fujikawa.
250 $a 2nd ed.
260 $a Cham : $b Springer International Publishing : $b Imprint: Springer, $c 2018.
300 $a xii, 215 p. : $b ill., digital ; $c 24 cm.
505 0 $a Introduction -- Excitotoxic programmed cell death involves caspase-independent mechanisms -- To survive or to die: how neurons deal with it -- Oxidative damage mechanisms in traumatic brain injury and antioxidant neuroprotective approaches -- Mitochondrial damage in traumatic CNS injury -- Neuroprotective agents target molecular mechanisms of programmed cell death after traumatic brain injury -- Involvement of apoptosis-inducing factor (AIF) in neuronal death following cerebral ischemia -- Apoptosis-inducing factor translocation to nuclei after transient global ischemia -- Necroptosis in cerebral ischemia -- Histological and elemental changes in ischemic stroke -- Hypoglycemic brain damage -- Activation of caspase-independent programmed pathways in seizure-induced neuronal necrosis -- Conclusion.
520 $a An overview of the biochemical mechanisms that produce acute nerve cell death in the brain. Covers injuries and disorders including stroke, brain and spinal cord trauma, hypoglycemic coma, and prolonged epileptic seizures. All of these lead to high concentrations of calcium in nerve cells which, in turn, causes degradation of cytoplasmic proteins, cleavage of nuclear DNA, and eventually cell death. The Second Edition contains 11 thoroughly updated chapters and 3 additional chapters that did not appear in the previous edition.
650 0 $a Nervous system $x Diseases $x Cytopathology. $3 1066203
650 0 $a Cell death. $3 665792
650 1 4 $a Biomedicine. $3 890831
650 2 4 $a Neurosciences. $3 588700
650 2 4 $a Neurology. $3 588698
650 2 4 $a Pathology. $3 643180
650 2 4 $a Cell Biology. $3 892383
650 2 4 $a Neurobiology. $3 588707
650 2 4 $a Psychiatry. $3 517037
700 1 $a Fujikawa, Denson G. $3 1066202
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856 4 0 $u http://dx.doi.org/10.1007/978-3-319-77495-4
950 $a Biomedical and Life Sciences (Springer-11642)