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The etiology of atopic dermatitis

Allen, Herbert B.

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The etiology of atopic dermatitis

紀錄類型:	書目-電子資源 : Monograph/item
正題名/作者:	The etiology of atopic dermatitis/ by Herbert B. Allen.
作者:	Allen, Herbert B.
出版者:	London :Springer London : : 2015.,
面頁冊數:	xviii, 117 p. :ill. (some col.), digital ;24 cm.
內容註:	Clinical presentations -- Microbiology -- Pathology -- Immunology -- Genetics -- Physiology -- Treatment -- Diseases in which eczema is a secondary component (Meyerson's nevus and Doucas Kapetanakis pigmented purpuric dermatosis) -- Diseases with occluded sweat ducts other than eczema (tinea pedis, axillary granular parakeratosis, seborrheic dermatitis) -- The Story of Eczema in Pictures -- Epilogue: A comparison of psoriasis and eczema: both caused by bacteria, but neither an infection.
Contained By:	Springer eBooks
標題:	Atopic dermatitis - Etiology. -
電子資源:	http://dx.doi.org/10.1007/978-1-4471-6545-3
ISBN:	9781447165453 (electronic bk.)

The etiology of atopic dermatitis
Allen, Herbert B.

The etiology of atopic dermatitis[electronic resource] /by Herbert B. Allen. - London :Springer London :2015. - xviii, 117 p. :ill. (some col.), digital ;24 cm.

Clinical presentations -- Microbiology -- Pathology -- Immunology -- Genetics -- Physiology -- Treatment -- Diseases in which eczema is a secondary component (Meyerson's nevus and Doucas Kapetanakis pigmented purpuric dermatosis) -- Diseases with occluded sweat ducts other than eczema (tinea pedis, axillary granular parakeratosis, seborrheic dermatitis) -- The Story of Eczema in Pictures -- Epilogue: A comparison of psoriasis and eczema: both caused by bacteria, but neither an infection.

Atopic dermatitis has been called the itch that rashes, and this book reveals what causes the itch. It presents completely new and unique findings in eczema: sweat ducts that become occluded with staphylococcal biofilms trigger the innate immune system with TLR2 receptor activity and this leads to production of the itching and inflammation in this disease. The Etiology ofAtopic Dermatitis details new concepts that bacterial biofilms occlude sweat ducts, trigger the innate immune system, and produce the lesions in atopic dermatitis. The author discusses the findings in terms of microbiology, pathology, immunology, genetics, physiology, treatment, diseases where eczema is considered a secondary component, and diseases not previously thought to be eczema, and followed by an epilogue where eczema and psoriasis are compared. Both these diseases are caused by bacteria, but neither one can be considered an infection. As such, this book is for all who take care of patients with atopic dermatitis, including dermatologists, pediatricians, family practice doctors, allergists and pathologists. It will also be interesting for those involved in research in microbiology, physiology, immunology, and genetics.

ISBN: 9781447165453 (electronic bk.)

Standard No.: 10.1007/978-1-4471-6545-3doiSubjects--Topical Terms:

2131276
Atopic dermatitis
--Etiology.

LC Class. No.: RL243

Dewey Class. No.: 616.51

The etiology of atopic dermatitis
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505 0 $a Clinical presentations -- Microbiology -- Pathology -- Immunology -- Genetics -- Physiology -- Treatment -- Diseases in which eczema is a secondary component (Meyerson's nevus and Doucas Kapetanakis pigmented purpuric dermatosis) -- Diseases with occluded sweat ducts other than eczema (tinea pedis, axillary granular parakeratosis, seborrheic dermatitis) -- The Story of Eczema in Pictures -- Epilogue: A comparison of psoriasis and eczema: both caused by bacteria, but neither an infection.
520 $a Atopic dermatitis has been called the itch that rashes, and this book reveals what causes the itch. It presents completely new and unique findings in eczema: sweat ducts that become occluded with staphylococcal biofilms trigger the innate immune system with TLR2 receptor activity and this leads to production of the itching and inflammation in this disease. The Etiology ofAtopic Dermatitis details new concepts that bacterial biofilms occlude sweat ducts, trigger the innate immune system, and produce the lesions in atopic dermatitis. The author discusses the findings in terms of microbiology, pathology, immunology, genetics, physiology, treatment, diseases where eczema is considered a secondary component, and diseases not previously thought to be eczema, and followed by an epilogue where eczema and psoriasis are compared. Both these diseases are caused by bacteria, but neither one can be considered an infection. As such, this book is for all who take care of patients with atopic dermatitis, including dermatologists, pediatricians, family practice doctors, allergists and pathologists. It will also be interesting for those involved in research in microbiology, physiology, immunology, and genetics.
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