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Resistance of Bacillus subtilis spor...

Xue, Yaming.

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Resistance of Bacillus subtilis spores lacking either nucleotide excision repair or spore photoproduct lyase to ultraviolet (UV) radiation from artificial or natural sources.

Record Type:	Language materials, printed : Monograph/item
Title/Author:	Resistance of Bacillus subtilis spores lacking either nucleotide excision repair or spore photoproduct lyase to ultraviolet (UV) radiation from artificial or natural sources./
Author:	Xue, Yaming.
Description:	57 p.
Notes:	Source: Masters Abstracts International, Volume: 35-06, page: 1727.
Contained By:	Masters Abstracts International35-06.
Subject:	Biology, Genetics. -
Online resource:	http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=1385346
ISBN:	9780591458176

Resistance of Bacillus subtilis spores lacking either nucleotide excision repair or spore photoproduct lyase to ultraviolet (UV) radiation from artificial or natural sources.
Xue, Yaming.

Resistance of Bacillus subtilis spores lacking either nucleotide excision repair or spore photoproduct lyase to ultraviolet (UV) radiation from artificial or natural sources. - 57 p.

Source: Masters Abstracts International, Volume: 35-06, page: 1727.

Thesis (M.S.)--University of North Texas Health Science Center at Fort Worth, 1996.

Exposure of bacterial spores to UV radiation causes the accumulation of a unique pyrimidine dimer in the DNA, "spore photoproduct" (SP). In Bacillus subtilis, two distinct DNA repair pathways are used for removal of SP: general nucleotide excision repair (the uvr pathway), or the SP-specific enzyme SP lyase (the spl pathway). Spores of four strains of Bacillus subtilis differing in their repair capabilities were irradiated under either artificial or solar UV. To determine the biologically-relevant cumulative UV dose under each irradiation condition, a sporocidal dosimeter was constructed. The results showed: (i) Both uvr and spl pathways contributed to the survival of spores under all tested conditions. The spl pathway was more efficient than uvr pathway in repairing the DNA damage caused by UV-C and solar UV-A, but no significant difference was noted in repairing DNA damage caused by UV-B or full-spectrum solar UV. (ii) Exposure of spores to solar UV can cause cellular lethal damage which is reparable by neither repair pathway.

ISBN: 9780591458176Subjects--Topical Terms:

1017730
Biology, Genetics.

Resistance of Bacillus subtilis spores lacking either nucleotide excision repair or spore photoproduct lyase to ultraviolet (UV) radiation from artificial or natural sources.
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020 $a 9780591458176
035 $a (MiAaPQ)AAI1385346
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100 1 $a Xue, Yaming. $3 2096631
245 1 0 $a Resistance of Bacillus subtilis spores lacking either nucleotide excision repair or spore photoproduct lyase to ultraviolet (UV) radiation from artificial or natural sources.
300 $a 57 p.
500 $a Source: Masters Abstracts International, Volume: 35-06, page: 1727.
500 $a Major Professor: Wayne Nicholson.
502 $a Thesis (M.S.)--University of North Texas Health Science Center at Fort Worth, 1996.
520 $a Exposure of bacterial spores to UV radiation causes the accumulation of a unique pyrimidine dimer in the DNA, "spore photoproduct" (SP). In Bacillus subtilis, two distinct DNA repair pathways are used for removal of SP: general nucleotide excision repair (the uvr pathway), or the SP-specific enzyme SP lyase (the spl pathway). Spores of four strains of Bacillus subtilis differing in their repair capabilities were irradiated under either artificial or solar UV. To determine the biologically-relevant cumulative UV dose under each irradiation condition, a sporocidal dosimeter was constructed. The results showed: (i) Both uvr and spl pathways contributed to the survival of spores under all tested conditions. The spl pathway was more efficient than uvr pathway in repairing the DNA damage caused by UV-C and solar UV-A, but no significant difference was noted in repairing DNA damage caused by UV-B or full-spectrum solar UV. (ii) Exposure of spores to solar UV can cause cellular lethal damage which is reparable by neither repair pathway.
590 $a School code: 1250.
650 4 $a Biology, Genetics. $3 1017730
650 4 $a Biology, Microbiology. $3 1017734
650 4 $a Biophysics, General. $3 1019105
690 $a 0369
690 $a 0410
690 $a 0786
710 2 $a University of North Texas Health Science Center at Fort Worth. $3 1021741
773 0 $t Masters Abstracts International $g 35-06.
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791 $a M.S.
792 $a 1996
793 $a English
856 4 0 $u http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=1385346