東華大學圖書館 |

語系: 繁體中文

說明(常見問題)

回圖書館首頁

手機版館藏查詢

登入

回首頁

切換: 標籤 | MARC模式 | ISBD

Role of endothelin in modulating chr...

Abram, Sean R.

FindBook

Google Book

Amazon

博客來

Role of endothelin in modulating chronic pressure-natriuresis and blood pressure regulation in response to changes in dietary sodium intake.

紀錄類型:	書目-電子資源 : Monograph/item
正題名/作者:	Role of endothelin in modulating chronic pressure-natriuresis and blood pressure regulation in response to changes in dietary sodium intake./
作者:	Abram, Sean R.
面頁冊數:	109 p.
附註:	Source: Dissertation Abstracts International, Volume: 65-06, Section: B, page: 2722.
Contained By:	Dissertation Abstracts International65-06B.
標題:	Biology, Animal Physiology. -
電子資源:	http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=3135855
ISBN:	0496830589

Role of endothelin in modulating chronic pressure-natriuresis and blood pressure regulation in response to changes in dietary sodium intake.
Abram, Sean R.

Role of endothelin in modulating chronic pressure-natriuresis and blood pressure regulation in response to changes in dietary sodium intake. - 109 p.

Source: Dissertation Abstracts International, Volume: 65-06, Section: B, page: 2722.

Thesis (Ph.D.)--The University of Mississippi Medical Center, 2004.

Since its discovery in 1988, much has been learned about the role endothelin (ET or ET-1) in the pathophysiology of hypertension. Aberrations in renal ET-1 expression, synthesis, and excretion are now commonly associated with the hypertensive st

ISBN: 0496830589Subjects--Topical Terms:

1017835
Biology, Animal Physiology.

Role of endothelin in modulating chronic pressure-natriuresis and blood pressure regulation in response to changes in dietary sodium intake.
LDR:01777nmm 2200325 4500 001 1845801
005 20051024140514.5
008 130614s2004 eng d
020 $a 0496830589
035 $a (UnM)AAI3135855
035 $a AAI3135855
040 $a UnM $c UnM
100 1 $a Abram, Sean R. $3 1933946
245 1 0 $a Role of endothelin in modulating chronic pressure-natriuresis and blood pressure regulation in response to changes in dietary sodium intake.
300 $a 109 p.
500 $a Source: Dissertation Abstracts International, Volume: 65-06, Section: B, page: 2722.
500 $a Adviser: John E. Hall.
502 $a Thesis (Ph.D.)--The University of Mississippi Medical Center, 2004.
520 $a Since its discovery in 1988, much has been learned about the role endothelin (ET or ET-1) in the pathophysiology of hypertension. Aberrations in renal ET-1 expression, synthesis, and excretion are now commonly associated with the hypertensive st
520 $a The overall goal of this dissertation was to test the central hypothesis that chronic blockade of ETB receptors causes salt-sensitive hypertension by impairing renal-pressure natriuresis. To achieve this goal, the ETB receptor was blocked using
520 $a The first important finding was the fact that the renal endothelin system is upregulated in response to increases in sodium intake. Secondly, blockade of the ETB receptors results in a rightward shift of the chronic pressure-natriuresis relation
590 $a School code: 0805.
650 4 $a Biology, Animal Physiology. $3 1017835
650 4 $a Health Sciences, Medicine and Surgery. $3 1017756
650 4 $a Biophysics, Medical. $3 1017681
650 4 $a Health Sciences, Pathology. $3 1017854
690 $a 0433
690 $a 0564
690 $a 0760
690 $a 0571
710 2 0 $a The University of Mississippi Medical Center. $3 1017852
773 0 $t Dissertation Abstracts International $g 65-06B.
790 1 0 $a Hall, John E., $e advisor
790 $a 0805
791 $a Ph.D.
792 $a 2004
856 4 0 $u http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=3135855