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Exercise mediated changes in periphe...
~
Campbell, Jonathan Edward.
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Exercise mediated changes in peripheral glucocorticoid exposure in the fructose-fed hamster.
Record Type:
Electronic resources : Monograph/item
Title/Author:
Exercise mediated changes in peripheral glucocorticoid exposure in the fructose-fed hamster./
Author:
Campbell, Jonathan Edward.
Description:
78 p.
Notes:
Source: Masters Abstracts International, Volume: 45-02, page: 0839.
Contained By:
Masters Abstracts International45-02.
Subject:
Biology, Animal Physiology. -
Online resource:
http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=MR19715
ISBN:
9780494197158
Exercise mediated changes in peripheral glucocorticoid exposure in the fructose-fed hamster.
Campbell, Jonathan Edward.
Exercise mediated changes in peripheral glucocorticoid exposure in the fructose-fed hamster.
- 78 p.
Source: Masters Abstracts International, Volume: 45-02, page: 0839.
Thesis (M.Sc.)--York University (Canada), 2006.
Glucocorticoids (GC) have been implicated in the development of central obesity and insulin resistance, two key characteristics of metabolic syndrome (MS). Recently, increased exposure to GC within insulin-sensitive tissues such as skeletal muscle, adipose, and liver have been associated with the development of the MS even if circulating concentrations of GC are normal. GC action is determined by the prereceptor enzyme 11-beta hydroxysteroid dehydrogenase type 1 (11
ISBN: 9780494197158Subjects--Topical Terms:
1017835
Biology, Animal Physiology.
Exercise mediated changes in peripheral glucocorticoid exposure in the fructose-fed hamster.
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Source: Masters Abstracts International, Volume: 45-02, page: 0839.
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Thesis (M.Sc.)--York University (Canada), 2006.
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Glucocorticoids (GC) have been implicated in the development of central obesity and insulin resistance, two key characteristics of metabolic syndrome (MS). Recently, increased exposure to GC within insulin-sensitive tissues such as skeletal muscle, adipose, and liver have been associated with the development of the MS even if circulating concentrations of GC are normal. GC action is determined by the prereceptor enzyme 11-beta hydroxysteroid dehydrogenase type 1 (11
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http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=MR19715
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