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Inhibition of pear fruit ripening by...
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Villalobos-Acuna, Max G.
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Inhibition of pear fruit ripening by 1-methylcyclopropene and recovery of ripening capacity.
Record Type:
Language materials, printed : Monograph/item
Title/Author:
Inhibition of pear fruit ripening by 1-methylcyclopropene and recovery of ripening capacity./
Author:
Villalobos-Acuna, Max G.
Description:
147 p.
Notes:
Source: Dissertation Abstracts International, Volume: 70-11, Section: B, page: 6627.
Contained By:
Dissertation Abstracts International70-11B.
Subject:
Agriculture, Horticulture. -
Online resource:
http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=3379633
ISBN:
9781109487336
Inhibition of pear fruit ripening by 1-methylcyclopropene and recovery of ripening capacity.
Villalobos-Acuna, Max G.
Inhibition of pear fruit ripening by 1-methylcyclopropene and recovery of ripening capacity.
- 147 p.
Source: Dissertation Abstracts International, Volume: 70-11, Section: B, page: 6627.
Thesis (Ph.D.)--University of California, Davis, 2009.
Preharvest and postharvest treatments of 1-MCP were studied in California 'Bartlett' pears. Preharvest applications of 1-methylcyclopropene (1-MCP) were tested at rates ranging from 0 to 100 mg/L in 2006 and 2007. Premature fruit drop, maturity, ripening-associated changes, and physiological disorders were studied in fruit harvested 7 to 21d after application and either ripened at 20°C immediately after harvest or after 3.5 to 6 m storage at -1°C. Overall, 1-MCP reduced the incidence of premature fruit drop when compared with the untreated fruit, but to a lesser extent than 1-naphthaleneacetic acid (NAA). 1-MCP had a stronger effect on retarding ripening-associated changes, including softening than on changes in fruit maturation on the tree (loss of firmness). 1-MCP's effect on ripening was lost if fruit remained on the tree for longer than 14 d after application or after 3.5 m cold storage, regardless of treatment concentration. Shorter storage times were not evaluated. A reduction of superficial and senescent scald and especially internal breakdown incidence after cold storage was observed in fruit treated with 1-MCP.
ISBN: 9781109487336Subjects--Topical Terms:
1017832
Agriculture, Horticulture.
Inhibition of pear fruit ripening by 1-methylcyclopropene and recovery of ripening capacity.
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147 p.
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Source: Dissertation Abstracts International, Volume: 70-11, Section: B, page: 6627.
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Adviser: Elizabeth J. Mitcham.
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Thesis (Ph.D.)--University of California, Davis, 2009.
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Preharvest and postharvest treatments of 1-MCP were studied in California 'Bartlett' pears. Preharvest applications of 1-methylcyclopropene (1-MCP) were tested at rates ranging from 0 to 100 mg/L in 2006 and 2007. Premature fruit drop, maturity, ripening-associated changes, and physiological disorders were studied in fruit harvested 7 to 21d after application and either ripened at 20°C immediately after harvest or after 3.5 to 6 m storage at -1°C. Overall, 1-MCP reduced the incidence of premature fruit drop when compared with the untreated fruit, but to a lesser extent than 1-naphthaleneacetic acid (NAA). 1-MCP had a stronger effect on retarding ripening-associated changes, including softening than on changes in fruit maturation on the tree (loss of firmness). 1-MCP's effect on ripening was lost if fruit remained on the tree for longer than 14 d after application or after 3.5 m cold storage, regardless of treatment concentration. Shorter storage times were not evaluated. A reduction of superficial and senescent scald and especially internal breakdown incidence after cold storage was observed in fruit treated with 1-MCP.
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The other component of the project involved using 1-MCP postharvest. Pears were treated with 0.3 muL L-1 1-MCP for 12 h at 20°C immediately after harvest in two seasons and to pear fruit of four maturities. 1-MCP decreased rates of softening, ethylene production, respiration, and yellow color development, and reduced incidence of scald and internal breakdown. Ripening recovery induced by cold storage of 1-MCP treated fruit depended on maturity and season and was associated with stimulated ethylene production, including 1-aminocyclopropene carboxylic acid synthase (ACS) activity, 1-aminocyclopropene carboxylic acid oxidase (ACO) activity, and transcript levels of genes associated with these enzymes.
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http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=3379633
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